The way DCA works is by fixating itself to the Pyruvate Dehydrogenase Kinase and blocking it's ability to regulate the Pyruvate Dehydrogenase Complex.
The COO- Acetate radical ion is the active part of the DCA. The chlorocarbon part, CCl2 is inert and cannot react with other chemicals in the cells. This definitely blocks the Kinase.
What is needed is a small molecule with a COOH part and an inert part but which is not toxic.
The toxicity of DCA comes from the chlorocarbon which breaks down and deionizes sodium channels in the neurons, causing neuropathy.
Also, metabolites of DCA causes Trichloroethylene and other sybstances which are themselves carcinogens, which are especially toxic for the liver because they accumulate there.
I propose that instead of using DCA, we could simply use Potassium Pyruvate.
Pyruvate has one double bonded oxygen and one methyl radical attached to the first carbon. Those two components are more reactive than chlorine but are still quite inert, enough for it to have an effect on the Kinase without the toxic effects of chlorocarbons.
The only toxicity coming from Potassium Pyruvate or Calcium Pyruvate would come from the Potassium and the Calcium itself. You cannot take too much of that without causing kidney, stomach and blood pressure problems.
So I propose to use Potassium Pyruvate instead of Potassium Dichloroacetate.
The effects will be the same but the toxicity will no longer be there.